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Fas) and their ligands Ligation of Fas leads to T-cell apoptosis via the caspase pathway, thereby ending the immune response Mutations in Clonal Deletion Pathways Mice whose T-cells do not express Fas, display expanded lymphocyte populations in their secondary lymphoid organs, leading to profound lymphadenopathy, significant self-reactivity (including many autoantibodies), and autoimmune damage to many organs (including kidneys) Rare human diseases, ALPS 1a and 1b, are caused by similar mutations in Fas or FasL respectively, also leading to lymphadenopathy and autoantibodies IL-2 affects the Fas pathway, and can eventually lead to AICD by increasing FasL expression IL-2 also helps attenuate immunity by downregulating survival molecules (e.g