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Prenatal exposure to polycyclic aromatic hydrocarbons (PAHs) augments neonatal hyperoxic lung injury: role of cytochrome P450 (CYP)1A1, 1A2, and 1B1
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Oral, injected, inhaled, topical, nasal, ophthalmic, and combined glucocorticoid exposure may suppress the hypothalamic-pituitary-adrenal axis
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