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Moreover, the differential effects of treatments on microgliosis, neuronal loss, synaptic plasticity, Tau pathology, and neuroinflammation suggest that modulating neuroinflammation, preserving synaptic function, and reducing neuronal loss could be key strategies in addressing cognitive decline in AD
Nutramedix
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found no significant difference in DNA methylation in the entorhinal cortex of patients with AD compared to control (Lashley et al., 2015)
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